Max Muscle’s Triple Whey Protein

July 23, 2009

Max Muscle Triple Whey Protein

Triple Whey Protein is a cost effective high quality protein powder. Triple Whey Protein contains Whey Protein Concentrate ,Whey Protein Concentrate, Whey Protein Isolate and L-Glycine. Most people don’t take enough of clean sources of protein Triple Whey Protein provides an easy solution. Triple Whey Protein contains immune boosting beta-lactoglobulins, alpha-lactalbumin, serum albumin,immunoglobulins, lactoperoxidase, lactoferrin, glycomacropeptides, and lysozymes.  L-Glycine helps produce Growth Hormone.  Triple Whey Protein is an economical protein filled with high quality growth factors to give you the muscle growth you are looking for.

Servings = 65 servings/1 scoops per serving

Directions = Mix one scoop in 6-10 ounces of water,juice,non-fat milk,soy milk or any cold beverage of your choice.You can drink it 2-3 times per day as an excellent protein supplement.Triple Whey Protein is instantized so you can stir it with a spoon or shake it without a blender.Try mixing one or two scoops with 8 to 12 oz of your favorite liquid in a blender with 1 cup of ice.Add fresh,frozen or canned fruit like strawberries,bananas and pineapple for a great way to get your daily fruit and make your shake taste even better.Add an extra scoop of Triple Whey Protein for a super protein shake that exceeds 50 grams of protein.

Ingredients = Whey Protein Concentrate ,Whey Protein Concentrate, Whey Protein Isolate, L-Glycine

available @ Max Muscle San Diego

These statements have not been evaluated by the FDA.   This product information is not intended to diagnose, treat, cure or prevent disease.


Max Muscle’s Grape Seed Extract

July 20, 2009

Grape Seed Extract

Grape seed extract is a major component of the Mediterranean diet. Our Grape seed extract contains Activin grape seed, a premium quality grape seed extract providing a rich source of biologically active flavonoids, including oligomeric proanthocyanidins. Grape seed extract acts as an anti-inflammatory that can help improve circulation. Oligomeric proanthocyanidins have very strong antioxidant activity. Grape seed contains the compound resveratrol which has been show in clinical studies to increase the median lifespan by 56% of fish studied.  Grape seed extract is a very powerful protective food that may have life extending properties.

Servings = 120-60 servings/1-2 daily

Directions = Take 1-2 tablets daily with food.

Ingredients = Activin® grape seed extract, Green Tea Extract, Lemon Bioflavoids, Trans-Resveratol, Cellulose

available @ Max Muscle San Diego

These statements have not been evaluated by the FDA.   This product information is not intended to diagnose, treat, cure or prevent disease.


Bruce Lipton – Biology of Perception 2 of 7: The Secrets of the Human Cell

June 10, 2009

The Secrets of the Human Cell – We are NOT controlled by our Genes

Humans and cells have all the same functions. Genes are NOT the Brain of the cell. You can take genes out of a cell and the cell will still live. Take our brain out of our bodies and we are dead. What is the brain of the cell and what are genes for?

The 50 – 90 Trillion cells in our body are like machines made out up of around 70,000 – 90,000 protein parts. The PROTEIN parts provide you with your body structure.

PROTEIN is made up of strings of AMINO ACIDS. The difference in the proteins is the sequence of the amino acids.

Where does life come from? The movement of Protein by an environmental signal [hormones, ect]. Proteins can change shape when different signals are sent to them creating movement and work.

Simple example is digestion = enzymes made up of protein ripping apart food.


HCG – Human Chorionic Gonadotropin Use After Anabolic Steroid Cycles for Bodybuilding

May 10, 2009

HCG – Human Chorionic Gonadotropin Use After Anabolic Steroid Cycles for Bodybuilding by George Spellwin

Human Chorionic Gonadotropin (hCG) is a peptide hormone that mimics the action of luteinizing hormone (LH). LH is the hormone that stimulates the testes to produce testosterone. (1) More specifically LH is the primary signal sent from the pituitary to the testes, which stimulates the leydig cells within the testes to produce testosterone.

When steroids are administered, LH levels rapidly decline. The absence of an LH signal from the pituitary causes the testes to stop producing testosterone, which causes rapid onset of testicular degeneration. The testicular degeneration begins with a reduction of leydig cell volume, and is then followed by rapid reductions in intra-testicular testosterone (ITT), peroxisomes, and Insulin-like factor 3 (INSL3) – All important bio-markers and factors for proper testicular function and testosterone production. (2-6,19) However, this degeneration can be prevented by a small maintenance dose of hCG ran throughout the cycle. Unfortunately, most steroid users have been engrained to believe that hCG should be used after a cycle, during PCT. Upon reviewing the science and basic endocrinology you will see that a faster and more complete recovery is possible if hCG is ran during a cycle.

Firstly, we must understand the clinical history of hCG to understand its purpose and its most efficient application. Many popular “steroid profiles” advocate using hCG at a dose of 2500-5000iu once or twice a week. These were the kind of dosages used in the historical (1960’s) hCG studies for hypogonadal men who had reduced testicular sensitivity due to prolonged LH deficiency. (21,22) A prolonged LH deficiency causes the testes to desensitize, requiring a higher hCG dose for ample stimulation. In men with normal LH levels and normal testicular sensitivity, the maximum increase of testosterone is seen from a dose of only 250iu, with minimal increases obtained from 500iu or even 5000iu. (2,11) (It appears the testes maximum secretion of testosterone is about 140% above their normal capacity.) (12-18) If you have allowed your testes to desensitize over the length of a typical steroid cycle, (8-16 weeks) then you would require a higher dose to elicit a response in an attempt to restore normal testicular size and function – but there is cost to this, and a high probability that you won’t regain full testicular function.

One term that is critical to understand is testosterone secretion capacity which is synonymous to testicular sensitivity. This is the amount of testosterone your testes can produce from any given LH or hCG stimulation. Therefore, if you have reduced testosterone secretion capacity (reduced testicular sensitivity), it will take more LH or hCG stimulation to produce the same result as if you had normal testosterone secretion capacity. If you reduce your testosterone secretion capacity too much, then no amount of LH or hCG stimulation will trigger normal testosterone production – and this leads to permanently reduced testosterone production.

To get an idea of how quickly you can reduce your testosterone secretion capacity from your average steroid cycle, consider this: LH levels are rapidly decreased by the 2nd day of steroid administration. (2,9,10) By shutting down the LH signal and allowing the testis to be non-functional over a 12-16 week period, leydig cell volume decreases 90%, ITT decreases 94%, INSL3 decreases 95%, while the capacity to secrete testosterone decreases as much as 98%. (2-6)

Note: visually analyzing testes size is a poor method of judging your actual testicular function, since testicular size is not directly related to the ability to secrete testosterone. (4) This is because the leydig cells, which are the primary sites of testosterone secretion, only make up about 10% of the total testicular volume. Therefore, when the testes may only appear 5-10% smaller, the testes ability to secrete testosterone upon LH or hCG stimulation can actually be significantly reduced to 98% of their normal production. (3-5) The point here is to not judge testosterone secretion capacity by testicular size.

The decreased testosterone secretion capacity caused by steroid use was well demonstrated in a study on power athletes who used steroids for 16 weeks, and were then administered 4500iu hCG post cycle. It was found that the steroid users were about 20 times less responsive to hCG, when compared to normal men who did not use steroids. (8) In other words, their testosterone secretion capacity was dramatically reduced because they did not receive an LH signal for 16 weeks. The testes essentially became desensitized and crippled. Case studies with steroid using patients show that aggressive long-term treatment with hCG at dosages as high as 10,000iu E3D for 12 weeks were unable to return full testicular size. (7) Another study with men using low dose steroids for 6 weeks showed unsuccessful return of Insulin-like factor-3 (INSL3) concentration in the testes upon 5000iu/wk of HCG treatment for 12 weeks (6) (INSL3 is an important biomarker for testosterone production potential and sperm production. 20)

These studies show that postponing hCG usage until the end of a steroid cycle increases your need for a higher dose of hCG, and decreases your odds of a full recovery. As a consequence to using a higher dose of hCG at the end of a cycle, estrogen will be increased disproportionately to testosterone, which then causes further HPTA suppression (from high estrogen) while increasing the risk of gyno. (11) For example, high doses of hCG have been found to raise estradiol up to 165%, while only raising testosterone 140%. (11) Higher doses of hCG are also known to reduce LH receptor concentration and degrade the enzymes responsible for testosterone synthesis within the testes (12,13,19 ) — the last thing someone wants during recovery. While these negative effects of hCG can be partly mitigated by the use of a SERM such as tamoxifen, it will create further problems associated with using a toxic SERM (covered in another article).

In light of the above evidence, it becomes obvious that we must take preventative measures to avoid this testicular degeneration. We must protect our testicular sensitivity. Besides, with hCG being so readily available, and such a painless shot, it makes you wonder why anyone wouldn’t use it on cycle.

Based on studies with normal men using steroids, 100iu HCG administered everyday was enough to preserve full testicular function and ITT levels, without causing desensitization typically associated with higher doses of hCG. (2) It is important that low-dose hCG is started before testicular sensitivity is reduced, which appears to rapidly manifest within the first 2-3 weeks of steroid use. Also, it’s important to discontinue the hCG before you start PCT so your leydig cells are given a chance to re-sensitize to your body’s own LH production. (To help further enhance testicular sensitivity, the dietary supplement Toco-8 may be used)

A more convenient alternative to the above recommendation would be a twice a week shot of 200iu hCG, or possibly a once a week shot of 500iu. However, it is most desirable to adhere to a lower more frequent dose of hCG to mimic the body’s natural LH release and minimize estrogen conversion. If you are starting hCG late in the cycle, one could calculate a rough estimate for their required hCG ‘kick starting’ dosage by multiplying 40iu x days of LH absence, since the testes will be desensitized, thus requiring a higher dose. (ie. 40iu x 60 days = 2400iu HCG dose)

Note: If following the on cycle hCG protocol, hCG should NOT be used for PCT.

For preservation of testicular sensitivity, use 100iu hCG ED starting 7 days after your first AAS dose. At the end of the cycle, drop the hCG two weeks before the AAS clear the system. For example, you would drop hCG about the same time as your last Testosterone Enanthate shot. Or, if you are ending the cycle with orals, you would drop the hCG about 10 days before your last oral dose. This will allow for a sudden and even clearance in hormone levels, while initiating LH and FSH production from the pituitary, to begin stimulating your testes to produce testosterone. Remember, recovery doesn’t begin until you are off hCG since your body will not release its own LH until the hCG has cleared the system.

In conclusion, we have learned that utilizing hCG during a steroid cycle will significantly prevent testicular degeneration. This helps create a seamless transition from “on cycle” to “off cycle” thus avoiding the post cycle crash.


Green Tea Improves Circulation

August 8, 2008

Green Tea improves blood circulation by being a vasodilator. Green Tea significantly decreases levels of the bad cholesterol, oxidized LDL. Green Tea also decreases the levels of an immune protective enzymes that fights oxidized LDL, called anti-oxidized LDL IgM antibodies.

Journal of the American College of Nutrition, Vol. 27, No. 2, 209-213 (2008)
Published by the American College of Nutrition

Green Tea Reduces LDL Oxidability and Improves Vascular Function

“This study found that consumption of green tea extract by women for five weeks produced modifications in vascular function and an important decrease in serum oxidizability.”


Phosphatidylserine Can Block Muscle Damage

July 31, 2008

Taking 600 mg of phosphatidylserine (PS) can lower the muscle damaging hormone cortisol by increasing the testosterone to cortisol ratio. The researchers suggest that phosphatidylserine is effective combating exercise-induced stress.

The effects of phosphatidylserine on endocrine response to moderate intensity exercise
Journal of the International Society of Sports Nutrition 2008, 5:11doi:10.1186/1550-2783-5-11

“The findings suggest that PS is an effective supplement for combating exercise-induced stress and preventing the physiological deterioration that can accompany too much exercise. PS supplementation promotes a desired hormonal status for athletes by blunting increases in cortisol levels.”

http://www.jissn.com/content/5/1/11


Stress Hormone Causes Aging

July 18, 2008

The stress hormone cortisol causes pre-mature aging of the immune system. Cortisol shortens a protective cap called telomere that allows the cells to keep dividing. The shorter the telomere, the shorter the cell’s lifespan will be. Telomeres shorten every time a cell divides the telomere shortens, acting like a clock for each cell.

The reason cancer cells are so strong is because their telomeres don’t shorten enough during cell division. A healthy cell lives a proper and preset time. An unhealthy cell either lives too short, not allowing the body to replace it time, or too long, allowing uncontrolled growth.

http://www.eurekalert.org/pub_releases/2008-07/uoc–usi071508.php